That is very cool but additionally a little bit complicated. My understanding is that, in contrast to people and some whale species, most mammals don’t truly undergo menopause. That means, most mammals (together with mice) don’t cease making key hormones like estradiol halfway via life. So, I’m curious whether or not this mouse mannequin of ovarian growing older is much like the human mannequin in that we truly run out of eggs (follicles), which is what causes menopause. So, if there aren’t any extra follicles available, one would assume that even one thing as nice as rapamycin wouldn’t have the ability to “flip again time” by creating extra follicles in people.
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Some extent about why slowing ovarian growing older (which rapamycin appears to do) is vital for girls in a broader context:
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@RapAdmin Thanks for sharing these articles. Fascinating to see the analysis that retains popping out. And inspiring as nicely. I’ve been taking rapamycin for about 10 weeks up to now and have seen that my cycle size has elevated by about 2 days the final two months – undoubtedly a very good factor for me. Can be attention-grabbing to see how the pattern continues.
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I’ve lined the current Longevity Summit convention right here, and the presentation by Jennifer Garrison and others associated to feminine reproductive well being.
See Right here: The Longevity Summit, Information & Replace – #13 by RapAdmin
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A brand new group targeted on funding analysis into bettering feminine reproductive longevity and well being:
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